Leptin and soluble leptin receptor in risk of colorectal cancer in the European Prospective Investigation into Cancer and Nutrition cohort.

نویسندگان

  • Krasimira Aleksandrova
  • Heiner Boeing
  • Mazda Jenab
  • H Bas Bueno-de-Mesquita
  • Eugene Jansen
  • Fränzel J B van Duijnhoven
  • Sabina Rinaldi
  • Veronika Fedirko
  • Isabelle Romieu
  • Elio Riboli
  • Marc J Gunter
  • Sabine Westphal
  • Kim Overvad
  • Anne Tjønneland
  • Jytte Halkjær
  • Antoine Racine
  • Marie-Christine Boutron-Ruault
  • Françoise Clavel-Chapelon
  • Rudolf Kaaks
  • Annekatrin Lukanova
  • Antonia Trichopoulou
  • Pagona Lagiou
  • Dimitrios Trichopoulos
  • Amalia Mattiello
  • Valeria Pala
  • Domenico Palli
  • Rosario Tumino
  • Paolo Vineis
  • Genevieve Buckland
  • María-José Sánchez
  • Pilar Amiano
  • José María Huerta
  • Aurelio Barricarte
  • Virginia Menéndez
  • Petra H Peeters
  • Stefan Söderberg
  • Richard Palmqvist
  • Naomi E Allen
  • Francesca L Crowe
  • Kay-Tee Khaw
  • Nickolas Wareham
  • Tobias Pischon
چکیده

Leptin, a peptide hormone produced primarily by the adipocytes, is hypothesized to play a role in the pathogenesis of colorectal cancer (CRC). Soluble leptin receptor (sOB-R) may regulate leptin's physiologic functions; however its relation to CRC risk is unknown. This study explored the association of leptin and sOB-R with risk of CRC in a prospective nested case-control study within the European Prospective Investigation into Cancer and Nutrition (EPIC) cohort. A total of 1,129 incident CRC cases (713 colon, 416 rectal) were matched within risk sets to 1,129 controls. Conditional logistic regression was used to calculate relative risks (RR) and 95% confidence intervals (CI). After multivariable adjustment including body mass index (BMI), waist circumference, and baseline leptin concentrations, sOB-R was strongly inversely associated with CRC (RR comparing the highest quintile vs. the lowest, 0.55; 95% CI, 0.40-0.76; P(trend) = 0.0004) and colon cancer (RR, 0.42; 95% CI, 0.28-0.63, P(trend) = 0.0001); whereas no association was seen for rectal cancer (RR adjusted for BMI and waist circumference, 0.83; 95% CI, 0.48-1.44, P(trend) = 0.38). In contrast, leptin was not associated with risk of CRC (RR adjusted for BMI and waist circumference, 0.85; 95% CI, 0.56-1.29, P(trend) = 0.23). Additional adjustments for circulating metabolic biomarkers did not attenuate these results. These novel findings suggest a strong inverse association between circulating sOB-R and CRC risk, independent of obesity measures, leptin concentrations, and other metabolic biomarkers. Further research is needed to confirm the potentially important role of sOB-R in CRC pathogenesis.

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عنوان ژورنال:
  • Cancer research

دوره 72 20  شماره 

صفحات  -

تاریخ انتشار 2012